Epidemiology


Etiology


Pathophysiology

Chromosomal nondisjunction → chromosome X monosomy/mosaicism → impaired ovarian development → malfunctioning streak gonads with connective tissue instead of normal germ cells → estrogen and progesterone deficiencies

  • Karyotype
    • Meiotic nondisjunction (most often in paternal gametes) → complete sex chromosomal monosomy (45,XO; no Barr body)
      • Barr body: The inactive X chromosome present in all female somatic cells. Appears as a small, dark-staining spot at the periphery of the nucleus. Consists of tightly-packed, transcriptionally-inactive, heterochromatin.
    • Mitotic nondisjunction of an embryonic cell → sex chromosomal mosaicism (45,XO/46,XX) → mild phenotypic expression

Clinical features

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  • Lymphatic system abnormalities
    • Cystic hygroma
      • a congenital lymphatic cyst (macrocystic lymphangioma) in the posterior triangle of the neck caused by malformation and obstruction of the fetal lymphatic system
      • Present at birth as a soft, compressible, painless, posterior triangle neck mass
      • Can cause dysphagia or airway compromise
    • Lymphedema of the hands and feet in the neonatal period
  • Musculoskeletal findings
    • Short stature: due to the presence of only one copy of the SHOX (short stature homeobox) gene, normally located on the X chromosome
    • Scoliosis are common
    • Shield chest: broad chest with widely spaced nipples
    • Webbed neck: skin folds along the side of the neck between the mastoid process and the acromion
    • Cubitus valgus
    • Short fourth metacarpals/metatarsals, nail dysplasia
    • High arched palate
    • Low-set posterior hairline
    • Osteoporosis and pathologic fractures
  • Cardiovascular abnormalities
  • Other disorders

Tip

Most patients with Turner syndrome have normal intelligence.


Diagnostics


Treatment