Epidemiology


Etiology


Pathophysiology


  • Rhabdomyolysis → release of the following substances:
    • Creatine phosphokinase (CPK) and serum myoglobin → pigment nephropathy → acute tubular necrosisacute kidney injury (intrinsic)
    • Potassium → cardiac arrhythmia
    • Lactic acid → metabolic acidosis
  • Hypovolemia → ↓ renal perfusion → acute kidney injury (prerenal)
    • Hypovolemia is predominantly due to third spacing of extracellular fluid into damaged muscle tissue.
  • Reperfusion syndrome → compartment syndrome

Clinical features


Diagnostics


  • Positive urine dipstick for blood without RBCs on microscopy

Treatment