• Amyloid: insoluble protein or protein fragments
  • Amyloidosis: extracellular aggregation and subsequent deposition of amyloid in various organs

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AL amyloidosis (primary amyloidosis)

Most common form of amyloidosis

Etiology

Low-level expansion of a plasma cell dyscrasia (e.g., multiple myeloma, Waldenstrom macroglobulinemia)


Pathophysiology

Increased production of the light chains of immunoglobulins → deposition of amyloid light chain protein (AL protein)


Clinical features


Diagnostics


Treatment


AA amyloidosis (secondary amyloidosis)

Etiology

AA amyloidosis is secondary to a chronic disease, such as:


Pathophysiology

Chronic inflammatory process → ↑ production of acute phase reactant SAA (serum amyloid-associated protein) → deposition of AA (amyloid-associated) protein in various organs


Clinical features

Tip

The main feature of AA amyloidosis at diagnosis is renal dysfunction (e.g., CKD, nephrotic syndrome). Cardiac involvement is rare.


Diagnostics

Tip

Amyloidosis should always be considered in patients who present with kidney, liver, or GI involvement in the setting of chronic inflammatory and/or infectious disease.


Treatment


ATTRmt amyloidosis

  • Autosomal dominant disease (most common) that is due to mutated transthyretin (ATTR)
  • Familial amyloid cardiomyopathy (FAC)
    • Common in African Americans (3% of African Americans carry the mutant allele for FAC)
    • Deposition in ventricular endomyocardium → restrictive cardiomyopathyL44816.jpg
    • Atrial deposition → arrhythmia