Hormonal contraception


  • Mechanism of Action
    • Hormonal contraceptives utilize synthetic hormones, typically a combination of estrogen and progestin, or progestin-only.
    • Estrogen Component (e.g., Ethinyl estradiol):
      • Suppresses FSH release from the pituitary via negative feedback.
      • This prevents the development of a dominant ovarian follicle, thus inhibiting ovulation.
    • Progestin Component (e.g., Levonorgestrel, Norethindrone, Drospirenone):
      • Suppresses LH surge, which is the primary trigger for ovulation.
      • Thickens cervical mucus, creating a barrier that prevents sperm penetration.
      • Thins the uterine endometrium, making it less suitable for implantation.
    • Progestin-only methods rely more heavily on thickening cervical mucus and thinning the endometrium, with ovulation inhibition being less consistent (especially with low-dose pills).
  • Types of Hormonal Contraception
    • Combined Hormonal Contraceptives (CHCs) - contain both estrogen and progestin.
      • Oral Contraceptive Pills (OCPs): Most common form, taken daily. Efficacy is >99% with perfect use, but closer to 91% with typical use.
      • Transdermal Patch: Applied weekly for 3 weeks, followed by a patch-free week.
      • Vaginal Ring: Inserted for 3 weeks, followed by a ring-free week.
    • Progestin-Only Contraceptives (POCs) - indicated for patients with contraindications to estrogen (e.g., smokers >35, history of VTE, migraine with aura).
      • Progestin-Only Pills (POPs or “minipill”): Must be taken at the same time each day due to lower hormone dose.
      • Injectable Contraception (e.g., Depo-Provera): Administered every 3 months. Associated with potential for delayed return to fertility and decreased bone mineral density with long-term use.
      • Subdermal Implant (e.g., Nexplanon): A small rod inserted in the upper arm, effective for 3 years. Highly effective (>99%) due to user independence.
      • Hormonal Intrauterine Device (IUD) (e.g., Mirena, Kyleena): T-shaped device placed in the uterus, effective for 5-8 years depending on the brand. Works primarily by local progestin effects on mucus and endometrium. Highly effective (>99%).
  • Key Side Effects & Adverse Events
    • Common Side Effects (often improve after 2-3 months):
      • Breakthrough bleeding/spotting (especially common with POPs and in the first few cycles of CHCs).
      • Headaches, nausea, breast tenderness, bloating.
      • Mood changes.
    • Serious Adverse Events (mainly associated with estrogen in CHCs):
      • Venous Thromboembolism (VTE): ↑ risk of DVT and PE. Risk is highest in women who smoke, are obese, or have thrombogenic mutations.
      • Hypertension: Estrogen can increase blood pressure.
      • Stroke & Myocardial Infarction: Risk is significantly elevated in women who smoke and are over age 35.
      • Hepatic Adenoma: A rare, benign liver tumor.
      • Gallbladder Disease: Increased risk of cholelithiasis.
  • Absolute Contraindications to Combined Hormonal Contraceptives
    • Hx of VTE (DVT/PE), stroke, or ischemic heart disease.
    • Smoker >35 years old (>15 cigarettes/day).
    • Severe/uncontrolled hypertension (>160/100 mmHg).
    • Migraine with aura (increased stroke risk).
    • Current breast cancer.
    • Active or severe liver disease (cirrhosis, liver tumor).
    • Known thrombogenic mutations (e.g., Factor V Leiden, Protein C/S deficiency).
    • <21 days postpartum due to increased VTE risk.
  • Non-Contraceptive Benefits (primarily CHCs)
    • Regulation of menstrual cycles, treatment for dysmenorrhea and menorrhagia.
    • Treatment for acne and hirsutism.
    • Management of symptoms related to PCOS and endometriosis.
    • ↓ risk of ovarian and endometrial cancer.
    • ↓ risk of benign breast disease.